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Ralph E Dolkart</span><span class="contrib-degrees">, M.D.</span>,</li><li><span class="name">Bernard Halpern</span><span class="contrib-degrees">, Ph.X.</span>and</li><li><span XXXXX="name">Janice Perlman</span><span class="contrib-XXXXXXX">, X.S.</span></li></ol> <h2>Abstract</XX> <p>XXX reason XXX the XXXXXXXXX XXXXXXXXX XXX XXXXXXXX of infections in diabetic XXXXXXXX has XXXXX XXXX satisfactorily XXXXXXXXX. This XXXXXXXXXXXXX XXX XXXXXXXX XX XXXXX XXX circulating antibody XXXXXXXX in alloxan diabetic, insulin treated diabetic XXX normal CF-1 XXXX injected XXXX XXXXXX XXXXX XXXXXXX. Only XXXXX XXXXXXX XXXXXXX XXXX alloxan XXX had elevated serum glucose XXXXXX (250 XX./XXX ml. or XXXXXX) XXXX included in XXX study, together with a group of normal animals. XXXXXXX XXXX XXXX XXXX XXX orbital XXXXX and the XXXXX XXXXXXXX for XXXXXXX binding capacity of BSA, glucose concentration and serum proteins. XXX was XXXXXXXXX XXXX I-XXX XXX XXX XXXXXXX binding XXXXXXXX of XXXX serum sample XXX XXXXXXXXXX as XXXXXXXXXX of BSA nitrogen bound XX X ml. of undiluted serum. XXX studies demonstrate that there XX no significant XXXXXXXXXX in XXXXXXXX response XX alloxan XXXXXXX XXXX, XXXXXXX XXXXXXX mice given XXXXXXX XXX normal XXXX when immunized XXXX BSA XXXXX XXX conditions of the XXXXXXXXXXX. XX XXX alloxan XXXXXXX XXXXX XXX be considered a laboratory model XX XXXXXXXX mellitus in XXX, XXX XXXXXXX described XXXXXX would be XXXXXXXXXX XXXX those studies of diabetics which have XXXXX no primary defect in their immune XXXXXXX.</p> <ul><li>XXXXXXXXX &XXXX; 1971 XX XXX XXXXXXXX Diabetes Association</li></ul>">
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